This is old news and not quite accurate. Y chromosomes are not needed to form testes since the SRY gene can be copied onto X chromosomes sometimes, which is how most males with XX chromosomes come into existence. FOXL2 and SOX9 causing gonad differentiation is accurate though. At this point in time scientists are basically working to make sure gender is fixed at birth in humans rather than it changing because it overwhelmingly looks like it can change in response to environmental pressures in humans. It looks like fat = make estrogens = turn female so you can make a baby, and no fat = no estrogens = turn male because there wouldn't be enough fat for the baby. We already see morphological changes and reduced fertility in humans under similar conditions, like fat men who grow breasts, have reduced sperm count and erectile dysfunction, and athletic women who stop menstruating, have their breasts shrink and their clitoris grow. Social policing seems to be what prevents people from actually crossing the sex line. There are already full morphological changes at puberty due to a gene that doesn't pop up in the article and doesn't relate to gonads e.g. in guevedoches though I hope that isn't begging for another explanation.

TL;DR I know the full process of sexual differentiation in humans, more or less, though it's knowledge that's actively discouraged from being sought.